Sympathetic Apraxia Symptoms

What are the Sympathetic Apraxia Symptoms?

The exclusive symptom of Sympathetic Apraxia is the paralysis of the right hand

This paralysis can be either as below

  • partial paralysis of the right hand
  • sometimes right hand might be fully paralysed

What is Apraxia?

Praxis, the ability to perform skilled or learned movements is essential for daily living.

Inability to perform such praxis movements is defined as apraxia.

Apraxia can be further classified into subtypes such as ideomotor, ideational and limb-kinetic apraxia. 

Apraxia is characterised by the below

  • loss of the ability to do certain learned movements, such as talk, walk, or understand tasks.

Praxic functions are frequently altered following brain lesion, giving rise to apraxia – a complex pattern of impairments that is difficult to assess or interpret.

Apraxia should be studied in consideration with and could contribute to other fields such as normal motor control, neuroimaging and neurophysiology.

What causes Apraxia?

Here are the causes of Apraxia

  • Damage to the Brain
  • Decrease in brain function which is called as degeneration of the brain, especially in areas of the brain that help to coordinate movements.

In patients with left hemispheric stroke, apraxia has been reported to be prevalent in approximately one-third of this population.

In clinical practice, it is not uncommon that more than one type of apraxia is present in a single affected patient

What is sympathetic apraxia?

Sympathetic apraxia is the inability of the non pathologic hand to carry out commanded movements.

Sympathetic apraxia is an ideomotor apraxia of the left hand.

Which condition is commonly associated with sympathetic apraxia?

Sympathetic apraxia is commonly associated with the below disorders

  • Right hemiparesis as well as
  • Broca’s aphasia

Where is the lesion underlying Sympathetic Apraxia? 

Sympathetic apraxia thought to be consequent upon frontal lobe lesions.

Sources

  1. Alexander MP, Benson DF, Stuss D: Frontal lobes and language. Brain Lang 37(4):656-691, 1989. 
  2. Benson DF: Aphasia, alexia and agraphia . New York: Churchill Livingstone, 1979. 
  3. Benson DF, Ardila A: Aphasia: a clinical perspective . New York: Oxford University Press, 1996. 
  4. Brazis PW, Masdeu JC, Biller J: Localization in clinical neurology, ed 6. Philadelphia: Lippincott Williams & Wilkins, 2011. 
  5. Cummings JL, Mega MS: Neuropsychiatry and behavioral neuroscience . New York: Oxford University Press, 2003. 
  6. Damasio AR, Damasio H: Aphasia and the neural basis of language. In Mesulam MM (ed): Principles of behavioral and cognitive neurology, ed 2. New York: Oxford University Press, pp. 294-315, 2000. 
  7. Feinberg TE, Farah MJ: Behavioral neurology and neuropsychology , ed 2 New York: McGraw Hill, 2003. 
  8. Freedman M, Alexander MP, Naeser MA: Anatomic basis of transcortical motor aphasias. Neurology 34(4):409-417l, 1984. 
  9. Mancall EL, Kirshner HS, et al.: Disorders of speech and language. Continuum Lifelong Learning Neurol 5(2):7-12, 1999. 
  10. Mesulam MM: Large scale neurocognitive networks and distributed processing for attention, language and memory. Ann Neurol 28(5):597-613, 1990. 
  11. Ross E: Affective prosody and the aprosodies. In Mesulam MM (ed): Principles of behavioral and cognitive neurology, ed 2. New York: Oxford University Press, pp. 316-331, 2000.
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