Sympathetic Apraxia Symptoms

What are the Sympathetic Apraxia Symptoms?

The exclusive symptom of Sympathetic Apraxia is the paralysis of the right hand

This paralysis can be either as below

  • partial paralysis of the right hand
  • sometimes right hand might be fully paralysed

What is sympathetic apraxia?

Sympathetic apraxia is the inability of the non pathologic hand to carry out commanded movements.

Sympathetic apraxia is an ideomotor apraxia of the left hand.

Which condition is commonly associated with sympathetic apraxia?

Sympathetic apraxia is commonly associated with the below disorders

  • Right hemiparesis as well as
  • Broca’s aphasia

Where is the lesion underlying Sympathetic Apraxia? 

Sympathetic apraxia thought to be consequent upon frontal lobe lesions.

What is Apraxia?

Praxis, the ability to perform skilled or learned movements is essential for daily living.

Inability to perform such praxis movements is defined as apraxia.

Apraxia can be further classified into subtypes such as ideomotor, ideational and limb-kinetic apraxia. 

Apraxia is a neurological condition characterized by the inability to execute or perform voluntary movements or gestures, despite having the physical ability and understanding to do so. It is a disorder of motor planning and coordination that affects the ability to carry out purposeful movements.

There are different types of apraxia, including:

  1. Ideomotor apraxia: This type of apraxia involves difficulty performing purposeful actions or movements upon command. Individuals with ideomotor apraxia may struggle to imitate gestures or use tools correctly.
  2. Ideational apraxia: Ideational apraxia affects the ability to conceptualize and plan a sequence of actions or tasks. It can make it challenging for individuals to perform complex activities that involve multiple steps or to use objects appropriately.
  3. Constructional apraxia: Constructional apraxia specifically affects visuospatial skills and the ability to copy or construct shapes, drawings, or structures. Individuals with constructional apraxia may have difficulty assembling puzzles, drawing simple shapes, or copying designs.

Apraxia can result from various causes, including stroke, brain injury, neurodegenerative disorders (such as Alzheimer’s disease), tumors, infections, or developmental conditions. It is typically diagnosed through a comprehensive neurological evaluation that assesses motor skills, coordination, and the ability to perform specific tasks.

Treatment for apraxia focuses on rehabilitation and compensatory strategies to help individuals regain or adapt their ability to perform daily activities. Occupational therapy, physical therapy, and speech therapy can play a crucial role in improving motor planning, coordination, and functional abilities.

What causes Apraxia?

What causes Sympathetic Apraxia?

It is caused by left frontal lesions disconnecting the left inferior parietal lobe from the right premotor cortex so that “praxicons” for the left hand cannot reach the hand area of the right frontal lobe. 

This condition is the apraxia of the left limb due to damage to the anterior left hemisphere.

This condition is most commonly associated with expressive aphasia.

Apraxia is characterised by the below

  • loss of the ability to do certain learned movements, such as talk, walk, or understand tasks.

Praxic functions are frequently altered following brain lesion, giving rise to apraxia – a complex pattern of impairments that is difficult to assess or interpret.

Apraxia should be studied in consideration with and could contribute to other fields such as normal motor control, neuroimaging and neurophysiology.

The exact cause of apraxia is not always clear, but it is believed to be related to disruptions in specific areas of the brain that are responsible for planning and coordinating movements. Several factors and conditions may contribute to the development of apraxia:

  1. Stroke: One of the most common causes of apraxia is a stroke, which occurs when there is a sudden disruption of blood flow to the brain, leading to damage in certain brain regions responsible for motor planning and execution.
  2. Brain Injury: Traumatic brain injuries resulting from accidents or falls can damage areas of the brain involved in motor control and contribute to apraxia.
  3. Degenerative Neurological Diseases: Certain neurodegenerative diseases, such as Alzheimer’s disease, frontotemporal dementia, and corticobasal degeneration, can cause apraxia as they progress and affect the brain’s motor areas.
  4. Brain Tumors: Tumors in the brain can compress or invade areas responsible for motor function and lead to apraxia.
  5. Neurological Disorders: Other neurological disorders, such as Parkinson’s disease, multiple sclerosis, and certain forms of epilepsy, can be associated with apraxia.
  6. Neurodevelopmental Disorders: In some cases, apraxia can be present from birth or early childhood and may be associated with neurodevelopmental disorders, such as developmental apraxia of speech (verbal apraxia) or developmental coordination disorder.
  7. Neurotransmitter Imbalances: Changes in the levels of neurotransmitters in the brain, such as dopamine or acetylcholine, can affect motor function and contribute to apraxia.
  8. Genetic Factors: Some forms of apraxia may have a genetic basis, and certain genetic mutations have been linked to the condition.

Sources

  1. Ideomotor Apraxia
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  4. Benson DF, Ardila A: Aphasia: a clinical perspective . New York: Oxford University Press, 1996. 
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  6. Cummings JL, Mega MS: Neuropsychiatry and behavioral neuroscience . New York: Oxford University Press, 2003. 
  7. Damasio AR, Damasio H: Aphasia and the neural basis of language. In Mesulam MM (ed): Principles of behavioral and cognitive neurology, ed 2. New York: Oxford University Press, pp. 294-315, 2000. 
  8. Feinberg TE, Farah MJ: Behavioral neurology and neuropsychology , ed 2 New York: McGraw Hill, 2003. 
  9. Freedman M, Alexander MP, Naeser MA: Anatomic basis of transcortical motor aphasias. Neurology 34(4):409-417l, 1984. 
  10. Mancall EL, Kirshner HS, et al.: Disorders of speech and language. Continuum Lifelong Learning Neurol 5(2):7-12, 1999. 
  11. Mesulam MM: Large scale neurocognitive networks and distributed processing for attention, language and memory. Ann Neurol 28(5):597-613, 1990. 
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